10/21/2017

Posterior Tibial Tendon Dysfunction (PTTD)

Posterior Tibial Tendon Dysfunction (PTTD)

Posterior tibial tendon dysfunction (PTTD), also known as posterior tibial tendonitis, is one of the leading causes of acquired flatfoot in adults. The onset of PTTD may be slow and progressive or sudden. An abrupt starting point is typically linked to some form of trauma, whether it be simple (stepping down off a curb or ladder) or severe (falling from a height or car accident). PTTD is hardly ever seen in children and increases in frequency with age.

The Characteristic Finding of PTTD Include;

Loss of medial arch height.

Edema (Swelling) of the Medial Ankle

Loss of the ability to resist force in order to abduct or push the foot out from the midline of the body.

Pain on the Medial Ankle With Weight Bearing

Inability to boost up on the foot without pain.

Too Many Toes Sign

Lateral subtalar joint (outside of the ankle) pain.

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Common test to evaluate PTTD could be the 'too many feet sign'. The way too many toes sign' is a test used to determine abduction deviation away from the midline of the body) with the forefoot. With damage to the rear tibial tendon, the forefoot will abduct or move out in relationship to the rest of the foot. In cases of PTTD, once the foot is viewed from at the rear of, the toes seem as 'too many' on the outside of the foot due to abduction of the forefoot.

Advanced cases of PTTD, in addition to the pain of the tendon itself, pain will also be noted at the sinus tarsi. The sinus tarsi refers to a small canal or divot on the outside of the ankle that can actually be felt. This tunnel is the entry to the subtalar joint. The subtalar joint is the joint that controls the side to side motion of the foot, motion that would occur with uneven surfaces or sloped hills. As PTTD progresses and the ability of the posterior tibial tendon to support the arch becomes declined, the arch will collapse overloading the subtalar joint. As a result, there is increased pressure applied to the joint areas of the lateral aspect of the subtalar joint, resulting in pain.

There have been many proposed explanations for PTTD over time because this condition was first described by Kulkowski inThe most contemporary explanation refers to an area of hypovascularity (limited blood flow) in the tendon just below the ankle. Tendon derives most of its' nutritional support from synovial fluid produced by the actual outer lining of the tendon. Really small blood vessels also permeate the tendon sheath to achieve tendons. This makes all tendon notoriously slow to be able to recover. In the case of the posterior tibial tendon, this problem is exacerbated by a distinct area of bad blood flow hypovascularity). This area is located in the posterior tibial tendon just below or distal to the inside ankle bone (medial malleolus).

Tendon is also the majority of susceptible to fatigue and failure at a place where the tendons changes direction. As the posterior tibial tendon descends the leg and comes to the inside of the ankle, the tendon follows a well defined groove in the back of the tibia (bone of the interior of the ankle). The tendon then takes a dramatic turn towards the arch of the foot. If the tendon is put into a situation where significant load is applied to the foot, the tendon responds by pulling up as the load of the body (in addition in order to gravity) pushes down. At the location where the tendon changes course, the tibia acts as a wedge and may utilize enough force to actually damage or rupture the tendon.

Equinus is Also a Contributing Factor to PTTD

Equinus is the term used to describe the ability or lack of ability to dorsiflex the feet on the ankle (move the toes toward you).Equinus is usually because of tightness in the leg muscle tissue, also known as the gastroc-soleal complex (a combination of the gastrocnemius and soleus muscles). Equinus may also be due to a bony block in the front of the ankle. The presence of equinus causes the rear tibial muscle to accept additional insert during gait.

Additional contributing factor to the onset of PTTD may include hypertension, diabetes, peripheral neuropathy, smoking or arthritis.

  • The progression of PTTD may well lead to tendonitis, partial tears of the tendon or perhaps complete tendon break.
  • Many classifications have been developed to describe PTTD.
  • The group as described by Johnson and Strom is most commonly used today.
  • Stage I Tendon status Attenuated (lengthened) with tendonitis but absolutely no rupture Clinical findings Palpable pain in the medial arch.
  • Foot will be supple, flexible with a lot of foot indicator X-ray/MRIMild to moderate tenosynovitis on MRI, no X-ray changes
  • Stage II Tendon status Attenuated with possible partial or complete shatter Clinical findings Pain in arch.
  • Can not raise on foot.
  • Too many toes indication present X-ray/MRI MRI notes tear in tendon.
  • X-ray noting abduction of forefoot, collapse of talo-navicular joint

Stage III Tendon status Severe degeneration with likely ruptureClinical findings Rigid flatfoot together with inability to raise up on toes X-ray/MRI MRI shows tear in tendon. X-ray observing abduction of forefoot, collapse of talo-navicular joint.

  • Treatment for PTTD is dependant after the clinical stage and the health status of the patient.
  • It is important to recognize thatPTTD is a mechanical problem that needs a mechanical solution.
  • This means that treating PTTD with medication alone is fraught with failure.
  • Timely introduction of some form of mechanised support is imperative.

Surgical procedures which usually focus on primary repair of the posterior tibial tendon happen to be very unsuccessful. This is due to the fact that muscle heals slowly following injuries and cannot be relied upon as a sole solution for PTTD cases. Surgical success is usually attained through stabilization of the rearfoot subtalar joint) which significantly reduces the work performed by the posterior tibial muscle.

Stage I May Respond to Sleep, Like a Walking Cast

Pain and inflammation may be controlled with anti-inflammatory medications. It is important to be sure that Stage I patients realize that the use of shoes with additional arch support and heel elevation, for the rest of their lives, is crucial. Arch support, whether built into the shoe or added as an orthotic, helps support the posterior tibial tendons and decrease its' work. Elevation of the heel, reduces equinus, one of the most significant contributing factors to PTTD. If Stage I patients go back to low heels without arch support, PTTD can recur.

Stage II patients, or Stage I patients that do not respond to rest and help, require surgical correction to be able to stabilize the subtalar joint prior to further damage to the posterior tibial tendon. Subtalar arthroeresis is a procedure used to strengthen the subtalar joint. Arthroeresis is a term that means the motion of the joint is blocked without fusion. Subtalar arthroeresis can only be used in cases of Stage I or II where mild to moderate deformation of the arch has occurred and MRI findings show the tendons to be only partially ruptured. Subtalar arthroeresis is typically performed in conjunction with anAchilles tendon lengthening procedure to correct equinus. These methods require casting for a period of weeks following the process.

Stage III patients require stabilization of the rearfoot with procedures that fuse the primary joints of the arch and base. These kinds of procedures are salvage procedures as well as require prolonged casting and disability following surgery. A common procedure forStage III is called triple arthrodesis which is a technique used to fuse the actual subtalar shared, the talo-navicular joint and the calcaneal cuboid joint.

PTTD is a condition that increases in frequency with age and the prevalence of poor health indicators such as diabetes and obesity. As a result, many patients with PTTD are bad surgical candidates for correction of PTTD. Prosthetics such as an ankle foot orthotic (AFO), Arizona Brace or other bracing may be very helpful to control the symptoms of PTTD. Anatomy:

The posterior tibial muscle is the extension of the posterior tibial muscle that lies deep to the leg. The origin of the rear tibial muscle is the posterior aspect of both the tibia and fibula and the interosseus membrane. The insertion of the posterior tibial muscle is the medial navicular where the tendon divides into nine different insertion site on the bottom of the foot.

Biomechanics:

The function of the posterior tibial tendon would be to plantarflex the feet at the toe away phase of the gait cycle and to support the medial arch.

Symptoms:

The symptoms of period I PTTD include a dull ache of the medial arch. The pain become worse with activity, better on days with limited time on the feet. Considerable activity may result in a partial rupture of the tendon, moving to stage II.

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  • Stage II symptoms are seen with more regularity.
  • Pain is present at the onset of walking and running.
  • Some constraint of a chance to raise up on the toes will be present.
  • Stage III signs are severe with an inability to accomplish most normal daily activities such as laundry washing or going to the store.
  • Collapse of the medial arch will be obvious.
  • Abduction of the forefoot will show 'too many toes sign'.

Differential Diagnosis:

Conditions that may resemble PTTD include tarsal tunnel syndrome, tibial stress fractures, posterior tibial muscle shatter, flexor hallucis longus tendonitis, gout, joint disease of the subtalar joint or a fracture of the posterior process of the actual talus.

Additional References Include;

Cantanzariti, A.R., Lee, M.S., Mendicino, R.W. PosteriorCalcaneal Displacement Osteotomy for Adult Acquired Flatfoot. J.of Foot and Ankle Surgery. 39-1: 2-14, 2000

  • Myerson, M.S., Corrigan, J.
  • Treatment of posterior tibial muscle dysfunction with flexor digitorum longus tendons transfer and calcaneal osteotomy.
  • Orthopedics 19:383-388, 1996

Myerson, M.S. Adult bought flatfoot deformity. J. Bone andJoint Surgery. 78-A;780, 1996

Johnson, K.A., Tibialis posterior tendon rupture. Clin. Orthop. 177:140-147, 1983

About the Actual Author:Jeffrey a

Oster, DPM, C.Ped is a board certified foot and ankle surgeon. Dr. Oster can also be board certified in pedorthics. Dr. Oster is medical director of Myfootshop.com and is in active practice in Granville, Ohio.

Domenic GoldenDomenic Golden
Domenic is a head content marketing specialist at musclenstress.com, a collection of articles on health issues. In the past, Domenic worked as a post curator for a well-known health site. When he's not writing posts, Domenic enjoys drawing and rock climbing.